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Analysis of genomic poly(A) sites
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 Citation for our APASdb
[1]. Leiming You, et al. APASdb: a database describing alternative poly(A) sites and selection of heterogeneous cleavage sites downstream of poly(A) signals. Nucleic Acids Res. 2015, 43: D59-D67.

Our Related Publication:
[0]. Xin Jia, Shaochun Yuan, Yao Wang, et al. The role of alternative polyadenylation in the antiviral innate immune response. Nat Commun. 2017, 8 :14605.

[1]. Guangrui Huang, et al. Dynamic regulation of tandem 3' untranslated regions in zebrafish spleen cells during immune response. J Immunol. 2016, 196: 715-725.

[2]. Yonggui Fu, Yutong Ge, Yu Sun, Jiahui Liang, et al. IVT-SAPAS: low-input and rapid method for sequencing alternative polyadenylation sites. PLoS One. 2015, 10: e0145477.

[3]. Jie Li, et al. Evaluation of two statistical methods provides insights into the complex patterns of alternative polyadenylation site switching. PLos One. 2015, 10: e0124324.

[4]. Peng Tian, Yu Sun, Yuxin Li, et al. A global analysis of tandem 3' UTRs in eosinophilic chronic rhinosinusitis with nasal polyps. PLos One. 2013, 7: e48997.

[5].Yuxin Li, Yu Sun, Yonggui Fu, et al. Dynamic landscape of tandem 3' UTRs during zebrafish development. Genome Res. 2012, 22: 1899-1906.

[6].Yu Sun, et al. Genome-wide alternative polyadenylation in animals: insights from high-throughput technologies. J Mol Cell Biol. 2012, 4: 352-361.

[7] Yonggui Fu, Yu Sun, Yuxin Li, et al. Differential genome-wide profiling of tandem 3′ UTRs among human breast cancer and normal cells by high-throughput sequencing. Genome Res. 2011, 10: 741-747.

May 27, 2016. last modified
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